gene expression omnibus geo Search Results


ncbi gene expression omnibus  (Fangman Specialties)
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Ncbi Gene Expression Omnibus, supplied by Fangman Specialties, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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gene expression omnibus  (Broad Institute Inc)
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Gene Expression Omnibus, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ncbi gene expression omnibus gse122314  (E McGrath)
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ncbi gene expression omnibus (geo)  (Hormel Health Labs)
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gene expression omnibus (geo) repository  (LabArchives LLC)
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ncbi gene expression omnibus  (MATHESON)
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data deposited in gene expression omnibus (geo) data sets or  (DNAnexus Inc)
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data deposited in gene expression omnibus (geo) data sets or - by Bioz Stars, 2026-08
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ncbi's gene expression omnibus  (ChaCha Search Inc)
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gse152859  (Ohta s Isan Co Ltd)
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gene expression omnibus dataset gse46170  (BioCarta)
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ncbi gene expression omnibus gse39621  (CH Instruments)
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cardiomyocyte mrna content ncbi gene expression omnibus gse119743  (Fenix GmbH)
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Fenix GmbH cardiomyocyte mrna content ncbi gene expression omnibus gse119743
( A ) Actin and myosin II stress fiber formation in non-muscle cells. Actin stress fibers are formed via the Arp2/3 complex and the formin mDia1. NMIIA is the predominant isoform at the leading edge of non-muscle cells, and stress fiber formation is NMIIA dependent. Non-muscle cells display robust retrograde flow of actin stress fibers and display rapid turnover. Large NMIIA stacks are formed via growth and expansion of smaller NMIIA filaments. Citations leading to this model are presented in the cartoon. ( B ) Model of actin and myosin II stress fiber formation in human <t>cardiomyocytes.</t> Sarcomeres are templated by Muscle Stress Fibers (MSFs). MSFs do not require the Arp2/3 complex, and require the formin FHOD3. MSFs display slow retrograde flow compared with non-muscle stress fibers. Both NMIIA and NMIIB are localized to the edge of hiCMs, and display prominent NMII co-filaments. NMIIB-βCMII co-filaments are also present with MSFs. Large βCMII filament stacks form via concatenation and stitching of individual βCMII filaments.
Cardiomyocyte Mrna Content Ncbi Gene Expression Omnibus Gse119743, supplied by Fenix GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


( A ) Actin and myosin II stress fiber formation in non-muscle cells. Actin stress fibers are formed via the Arp2/3 complex and the formin mDia1. NMIIA is the predominant isoform at the leading edge of non-muscle cells, and stress fiber formation is NMIIA dependent. Non-muscle cells display robust retrograde flow of actin stress fibers and display rapid turnover. Large NMIIA stacks are formed via growth and expansion of smaller NMIIA filaments. Citations leading to this model are presented in the cartoon. ( B ) Model of actin and myosin II stress fiber formation in human cardiomyocytes. Sarcomeres are templated by Muscle Stress Fibers (MSFs). MSFs do not require the Arp2/3 complex, and require the formin FHOD3. MSFs display slow retrograde flow compared with non-muscle stress fibers. Both NMIIA and NMIIB are localized to the edge of hiCMs, and display prominent NMII co-filaments. NMIIB-βCMII co-filaments are also present with MSFs. Large βCMII filament stacks form via concatenation and stitching of individual βCMII filaments.

Journal: eLife

Article Title: Muscle-specific stress fibers give rise to sarcomeres in cardiomyocytes

doi: 10.7554/eLife.42144

Figure Lengend Snippet: ( A ) Actin and myosin II stress fiber formation in non-muscle cells. Actin stress fibers are formed via the Arp2/3 complex and the formin mDia1. NMIIA is the predominant isoform at the leading edge of non-muscle cells, and stress fiber formation is NMIIA dependent. Non-muscle cells display robust retrograde flow of actin stress fibers and display rapid turnover. Large NMIIA stacks are formed via growth and expansion of smaller NMIIA filaments. Citations leading to this model are presented in the cartoon. ( B ) Model of actin and myosin II stress fiber formation in human cardiomyocytes. Sarcomeres are templated by Muscle Stress Fibers (MSFs). MSFs do not require the Arp2/3 complex, and require the formin FHOD3. MSFs display slow retrograde flow compared with non-muscle stress fibers. Both NMIIA and NMIIB are localized to the edge of hiCMs, and display prominent NMII co-filaments. NMIIB-βCMII co-filaments are also present with MSFs. Large βCMII filament stacks form via concatenation and stitching of individual βCMII filaments.

Article Snippet: The following dataset was generated: Fenix AM Burnette DT 2018 Cardiomyocyte mRNA Content NCBI Gene Expression Omnibus GSE119743

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